வாய் புற்றுநோய்

Based on the recently identified mutations in HNSCCs, the major pathologic pathways implicated in the tumorigenesis of HNSCC include dysregulation of four processes: 1. cellular survival and proliferation (e.g., TP53, EGFR, MET, and PIK3CA); 2. cell-cycle control (e.g., CDKN2A and CCND1); 3. cellular differentiation (e.g., NOTCH1); and 4. Adhesion and invasion signaling (e.g., FAT1).7 TP53, EGFR, PIK3CA, CDKN2A, CCND1, and MET participate in several common signaling pathways. Alterations of these genes are most frequently seen in alcohol and tobacco-related HNSCC. However their role in progn…

ஸ்பான்சர்: Banaras Hindu University
ClinicalTrials.gov-ல் NCT05382585
பதிவேட்டில் கடைசியாக 13 Aug 2024 அன்று புதுப்பிக்கப்பட்டது · நாங்கள் 20 Oct 2026 அன்று பார்த்தோம்